Sunday, June 10, 2007
Why did this happen?
This is where they tell me the almighty dollar comes into the picture. You see the BAD MAN that made me, did it for the money that he got for selling me. I came from a PUPPY MILL in Missouri. If you look on the front of the web blog and see the little photos of the dogs in cages, click on them and get some tissues out. They are YOU TUBE videos of places where my mom and dad might have been kept. You can also click on the left of the page that says PUPPY MILLS read the page and watch those videos too. Thats what a puppy mill is. Pretty disgusting huh? The BAD MAN that made me told the uneducated buyers that I was a MINIATURE bulldog. They believed him and bought me for a lot of money. Again people PLEASE listen up!!! THERE IS NO SUCH THING AS A MINIATURE BULLDOG!!!!! The BAD PEOPLE will only tell you that so you will give them money and then they will ship us in a plane and when we get there we will be sick!!! Just like me!!! I have been sick since the day I came to Washington state from Missouri.
My Diagnosis....
My diagnosis is not a good one. They thought at first I had congestive heart failure because I had so much edema around my heart on the xray. Dr Kramer sent a report to Dr Pam and this is what it said.
STUDY: Thorax
Current radiographs are dated 5/29 and are compared with four prior studies, most recently dated 5/24. The trachea is severely hypoplastic. There are dense ventrally distributed alveolar infiltrates. Limited improvement in the magnitude of these infiltrates is noted in the post-diuretic radiograph. Air bronchogram formation is noted in the cranial and middle lung fields. Bronchial wall thickening is noted caudodorsally. Cardiac volume probably is normal Liver volume is age-appropriate. Mildly prominent and possibly thickened rugae are noted in the gastric body region. The T7 vertebra is mildly anomalous.
ASSESSMENT:
1. Moderately consolidated ventrally distributed pneumonia infiltrates
2. Limited improvement after initiation of diuretic therapy
3. Chronic bronchitis
4. Severely hypoplastic trachea
COMMENTS:
Recurrent pneumonia is assumed although a component of acute respiratory distress syndrome also is possible. Ventilatory efforts are undoubtedly increased by the narrowed tracheal lumen. Any seasonal temperature elevations and resultant increased respiratory effort to dissipate heat will further worsen respiratory distress. Aggressive antimicrobial therapy is indicated and more specific evaluation such as bronchoscopy and endobronchial lavage should be considered, if the patient will tolerate invasive testing. Repeated episodes of respiratory diestess should be anticipated, given the very small tracheal cross-sectional area and the pulmonary injury that likely has accrued to date. Long-term antibiotic therapy will probably be required, and ancillary use of physiotherapy such as nebulization and coupage is probably also prudent. Internal medicine consultation is encouraged. The attendant expense and effort that likely will accompany repeated episodes of respiratory disease in this patient should be discussed carefully with the guradians of this patient.
So basically what this all means is if I am not monitored properly and not cared for properly I will die, pure and simple.
STUDY: Thorax
Current radiographs are dated 5/29 and are compared with four prior studies, most recently dated 5/24. The trachea is severely hypoplastic. There are dense ventrally distributed alveolar infiltrates. Limited improvement in the magnitude of these infiltrates is noted in the post-diuretic radiograph. Air bronchogram formation is noted in the cranial and middle lung fields. Bronchial wall thickening is noted caudodorsally. Cardiac volume probably is normal Liver volume is age-appropriate. Mildly prominent and possibly thickened rugae are noted in the gastric body region. The T7 vertebra is mildly anomalous.
ASSESSMENT:
1. Moderately consolidated ventrally distributed pneumonia infiltrates
2. Limited improvement after initiation of diuretic therapy
3. Chronic bronchitis
4. Severely hypoplastic trachea
COMMENTS:
Recurrent pneumonia is assumed although a component of acute respiratory distress syndrome also is possible. Ventilatory efforts are undoubtedly increased by the narrowed tracheal lumen. Any seasonal temperature elevations and resultant increased respiratory effort to dissipate heat will further worsen respiratory distress. Aggressive antimicrobial therapy is indicated and more specific evaluation such as bronchoscopy and endobronchial lavage should be considered, if the patient will tolerate invasive testing. Repeated episodes of respiratory diestess should be anticipated, given the very small tracheal cross-sectional area and the pulmonary injury that likely has accrued to date. Long-term antibiotic therapy will probably be required, and ancillary use of physiotherapy such as nebulization and coupage is probably also prudent. Internal medicine consultation is encouraged. The attendant expense and effort that likely will accompany repeated episodes of respiratory disease in this patient should be discussed carefully with the guradians of this patient.
So basically what this all means is if I am not monitored properly and not cared for properly I will die, pure and simple.
